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Furthermore, LDL receptors become dysfunctional, increasing serum LDL-C, while inhibition of HDL-C concentration in the blood causes undesirable changes in lipid metabolism, producing hypercholesterolemia (Yousef et al

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It remains to be established if free radicals are involved in the pathogenesis of glutathione synthetase deficiency and if patients with low levels of glutathione are more sensitive to oxidative stress

Within this framework, our observation of reduced thalamic GSH during the interictal phase may be regarded as a candidate neurochemical correlate of these chronic, between-attack thalamic changes, possibly reflecting cumulative oxidative burden in a region chronically exposed to trigeminovascular input over years of recurrent attacks

8: F1000 Faculty Rev-529

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