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IPF is a disease characterized by the progressive loss of lung function distinguished by severe pulmonary epithelial injury, fibroblast activation, extracellular matrix deposition, and distorted lung development

Superoxide dismutases (SOD) three forms exist, SOD1 (Intracellular), SOD2/MnSOD (Mitochondrial), SOD3 (Extracellular) Catalase (CAT) Glutathione peroxidase (GPx) In addition to these enzymes, glutathione transferase, ceruloplasmin, hemoxygenase and possibly several other enzymes may participate in enzymatic control of oxygen radicals and their products

central nervous system (CNS) axons barely regenerate at all after injury

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The mild acidity stabilizes these specific molecular structures