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Atherosclerosis serves as the primary pathological mechanism behind most cardiovascular disease (CVD) cases, a leading global health concern and one of the top causes of disability and premature death [444, 445]

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The complexity of its regulation, its context-dependent role in disease progression, and the challenge of achieving cell-type and disease-stage specificity necessitate a deeper mechanistic understanding

Mitochondrial biogenesis and proteome remodeling promote one-carbon metabolism for T cell activation

Prevented the gradual increase in fasting blood glucose levels seen in the high-selenium alone group, with levels remaining near those of the adequate-selenium control group by the fifth month