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Its function has been demonstrated in in vitro glycine uptake assays with reconstituted proteoliposomes (Lunetti et al., 2016), and in vivo , in zebrafish mutants ( SLC25A38 gene are a cause of transfusion-dependent congenital sideroblastic anemia ( SLC25A38 mutations are refractory to pyridoxine treatment, which is used to treat sideroblastic anemias caused by ALAS2 mutations that occur in the PLP binding region ( SLC25A38 mutations, these patients often have other disorders

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In a previous study, the clearance of CaP nanoparticles loaded with a mimetic peptide (MP) was delayed in the heart 9

In rats, enzymatic shedding of the glycocalyx is primarily induced by MMPs [158, 159]