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These observations imply a potential link between GHRP-6 exposure and increased GH levels

It is present at millimolar concentrations intracellularly and performs a foundational role in thiol-based redox signaling, protein function, gene expression, cell differentiation, and cell proliferation

The pathogenesis of GBM The pathogenesis of GBM includes gene mutations, signaling pathway dysregulation, epigenetic modifications, and tumor microenvironment (TME), which collectively drive tumor initiation, progression, invasion, and treatment resistance [13, 14]

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