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Arnold HH, Heinze H (1990)

3 Importantly, this study clarified a mechanistic subtlety often garbled in marketing copy: KPVs intestinal anti-inflammatory effect in that model was not mediated through melanocortin receptors but through PepT1-dependent uptake and downstream inhibition of NF-B signaling

Furthermore, our findings could reinforce the previously proven concept that AcM does not hydrolyze PGN of live bacteria, whilst leading to the indirect modulation of the microbiota, driven by the reduced protein availability in the caeca, with some measurable effects on the gene encoding PGN synthesis

Deng, Z

[4] References M