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There is therefore an urgent need to identify the subtype-specific molecular mechanisms underlying TNBC progression and resistance to chemotherapy that may represent therapeutic targets

Milunsky, Teratogenicity of High Vitamin A Intake, N

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Sympatholysis enhances the release of local endogenous, short-lived vasodilators such as adenosine, ATP, prostaglandins, prostacyclin, bradykinin, and protons to act as a compensatory mechanism (58)

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