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Though apparently these results might contradict the results obtained in HT22 cells, the outcomes suggest that it is under a low DHA diet with limited availability of n-3 precursors, when it is especially important to activate mechanisms (like upregulation of Gpx4 isoform expression) that may ensure the preservation of DHA in brain membranes

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The sustained receptor activation achieved by semaglutide produces more pronounced and consistent metabolic effects compared to the pulsatile, meal-related actions of endogenous GLP-1