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As demonstrated in Fig
Evidence that compromised K + spatial buffering contributes to the epileptogenic effect of mutations in the human K ir 4.1 gene (KCNJ10)
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The most commonly accepted theory is that a defect in the glycosaminoglycan layer of the bladder urothelium allows solutes in the urine to penetrate through the urothelium into the bladder interstitium which causes an inflammatory reaction [1]
Modulation of Hormones DSIP has been linked to increased production of growth hormone (GH) during sleep and may suppress corticotropin (ACTH) and cortisol , aligning with its stress-relieving effects
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