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Dinkla S, Wessels K, Verdurmen WP, Tomelleri C, Cluitmans JC, Fransen J, et al

Much of our understanding of the pathobiology of TBI has arisen from animal models that mimic features of human TBI

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This suggests that the activation of insulitis, which underpins the initiation and progression of diabetes, requires the immune tolerance to -cell autoantigens to be broken (1, 30)

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