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To test whether the observed p53 and apoptotic induction upon nutrient withdrawal is cell-autonomous, we employed in vitro adipocyte models

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Limitations of the model This model was designed to allow us to study various regulatory mechanisms in the transsulfuration pathway and the effects of oxidative stress, particularly as applied to Down syndrome and autism

Glutathionuria: inborn error of metabolism due to tissue deficiency of gamma-glutamyl transpeptidase

10.1007/s12016-015-8525-4 171 ResiliacJ.GraysonM