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Conversely, exogenous MSC-Exos have emerged as promising therapeutic tools to suppress ferroptosis in diverse diseases, as will be discussed in the later sections

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These findings suggest that the pathological process of SUI may involve TGF-/Smad3 signalling

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Mitophagy, a selective form of autophagy, specifically targets damaged mitochondria for degradation, thereby preventing the release of ROS and subsequent cellular damage