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Common side effects include nausea, constipation, and fatigue, particularly during dose escalation

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369 Advanced glycation end products, which are generated by glucose in a non-enzymatic manner, also promote ROS production and endoplasmic reticulum stress, and then induce insulin suppression via the protein kinase RNA-like endoplasmic reticulum kinase/forkhead box O1 pathway

Glymphatic Stasis TBI disrupts the polarization of Aquaporin-4 (AQP4) channels, leading to "glymphatic stasis." The brain cannot clear its own waste, leading to the accumulation of toxic proteins (Tau, Amyloid) [7]