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4 Impaired excretion is most often due to abnormalities in the kidney urate transporter (called URAT1) or organic ion transporter (OAT), both of which control the movement of uric acid out of proximal kidney tubules and into urine

Table 2), and this was improved following treatment with Fer-1 (26.81 2.71 g

2016;215:68.e168.e4

To elucidate the potential impact of TTR amyloidosisassociated oxidative stress on GSH homeostasis, we quantified plasma PGA levels in three well-defined cohorts aged 55 years and older: asymptomatic carriers of the TTR-V30M mutation, symptomatic carriers with confirmed TTR amyloidosis (TTR-V30M patients), and healthy controls with the wild-type TTR genotype

[DOI] [PMC free article] [PubMed] [Google Scholar] 133.liu S., Li T., Liu H., et al

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