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The appearance of drusen, typically following the formation of BLamD, further disrupts RPE organization, placing both mechanical and physiological stress on the surrounding RPE cells, which results in cell death [29]

The expression levels for each of the genes were normalized to fragments per kilobase of exon model per million mapped reads (FPKM) using a software package called RSEM 79

Yet, the body's response can vary widely

The resulting autophagic dysfunction has diverse effects on renal cell types, including tubular epithelial cells, podocytes, and glomerular endothelial cells

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